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“the microglia are supposed to be, from the get-go, constantly clearing amyloid-beta, as well as keeping a lid on inflammation,” added dr andreasson.
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Now stanford is hoping to produce a compound which only blocks ep2 to prevent unnecessary side effects.
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When they spot trouble, they release substances that recruit other microglia to the scene which then destroy and get rid of any foreign invaders dating now ep2.
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” the scientists discovered that in young mice, the microglia kept the sticky plaques under control.
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A-beta builds up in the brain, inducing toxic inflammation.
They act as a frontline defence, looking for suspicious activities and materials.
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Related articles “microglia are the brain’s beat cops,” said dr katrin andreasson, professor of neurology and neurological sciences at stanford university school of medicine.
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The researchers found that, in mice, blocking ep2 with a drug reversed memory loss and myriad other alzheimer’s-like features in the animals.
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Similarly mice which were genetically engineered not to have ep2 did not develop alzheimer s disease, even when injected with a solution of amyloid-beta, suggesting that their cells were getting rid of the protein naturally.
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Now scientists have shown that blocking the protein allows the microglia to function normally again so they can hoover up the dangerous sticky amyloid-beta plaques which damage nerve cells in alzheimer s disease.
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“if they lose their ability to function, things get out of control.
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They also work as garbage collectors, chewing up dead cells and molecular debris strewn among living cells including clusters of amyloid-beta which aggregate as gummy deposits and break the connections between neurons, causing loss of memory and spatial awareness.
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” by 2015 there will be 850,000 people with dementia in the uk, with alzheimer s disease being the most common type.
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Microglial cells make up around 10 to 15 per cent of cells in the brain.
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But when experiments were done on older mice, the protein ep2 swung into action and stopped the microglia producing enzymes which digested the plaques.
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Researchers at stanford university discovered that nerve cells die because cells which are supposed to clear the brain of bacteria, viruses and dangerous deposits, stop working.
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“our experiments show that keeping them on the right track counters memory loss and preserves healthy brain physiology.
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These clusters are believed to play a substantial role in causing alzheimer’s.